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In individuals with obesity, the balance of the signalling adipokines is considerably interrupted (Reference Sirico, Bianco and DAlicandro8)

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This commonly includes lifelong treatment for pernicious anaemia, managing severe B12 deficiency with neurological symptoms to prevent further damage, and for individuals post-gastric surgery due to impaired absorption

The most affected molecules were total glucosinolates, total indolyl, total aliphatic glucosinolates, and all individual glucosinolate levels (with the exception of 4-methoxyglucobrassicin)