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Hypertension in peripheral arterial disease

An emerging body of evidence supports the impact of SCFAs on improving glucose homeostasis, predominantly signaled through GPCRs, mainly GPR43 (FFAR2) and GPR41 (FFAR3) (31, 33)

Recently, it has been reported that an essential role in the pathogenesis of AD may be also played by other proteins such as -synuclein and the protein TDP-43 [7]