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Description

Control of cell cycle and immortality TERT TPMs are genetic alterations which mediate cancer cell immortalization by activating telomerase expression

Given the upregulation of compensatory metabolic pathways, including FSP1 and DHODH, in resistant glioblastoma cells, combinatorial inhibition of GPX4 and FSP1 represents a promising therapeutic avenue that warrants further investigation

Indeed, in recent years, different formulations of rHDL which employ synthetic apo A-I mimetics instead of naturally occurring apo A-I have been developed