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According to the results of another study, we reasonably hypothesize that the protective mechanism against chemotherapy-induced neurotoxicity may be that protective drugs and their metabolites increase the DNA-binding activity of Nrf2, thereby enhancing the ability to counteract oxidative stress (Kawashiri et al., 2018)

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Samples were run in triplicate on a RealTime Thermal Cycler (BioRad Laboratories), and variation was calculated using the C t method with respective endogenous controls