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Linnebank M, Moskau S, Semmler A, Widman G, Stoffel-Wagner B, Weller M, et al

[DOI] [PMC free article] [PubMed] [Google Scholar] Boutry-Regard C

For example, -KG supplementation can promote the transition of macrophages from M1 to M2/reparative phenotypes and improve disease activity in colitis models (123), providing support for the clinical research hypothesis of nutrition-metabolic anaplerosis-immune repair. Lowering circulating BCAA levels can inhibit M1 polarization, and modulation of their metabolic enzymes has become a therapeutic strategy in patients with obesity and insulin resistance (124)