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127 In mitochondria, ROS impact the TCA cycle by oxidizing enzymes such as aconitase and -ketoglutarate dehydrogenase (KGDH), modulating metabolic flux, whereas superoxide generated at the ETC amplifies redox signaling

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It is therefore not surprising the role of ROS either as apoptotic molecules or stimulators of cell proliferation, depending on the cell type and on the intensity of the stress produced

Loss of DJ-1 does not affect mitochondrial respiration but increases ROS production and mitochondrial permeability transition pore opening