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This review further summarized recent advances on research into the molecular mechanism of GH-IGF1 signaling pathway in these effects, expecting to specify the dual characters of GH-IGF1 signaling pathways in radiotherapy and post-radiotherapy repair of cancers, subsequently providing theoretical basis of their roles in increasing radiation sensitivity during cancer radiotherapy and repairing damage after radiotherapy

NAD+ (Nicotinamide Adenine Dinucleotide): Boosts cellular energy and repair

These findings highlight the potential of polyphenols as GSK3 inhibitors, offering a promising therapeutic strategy to modulate tau pathology and mitigate AD progression (Figure 5ah)