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3.2.6 Autophagy and MI Endothelial cell dysfunction initiates atherosclerotic plaque formation, and the rapid progression of unstable plaques is crucial to MI development (Vion et al., 2017)

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They observed that the treatments increased superoxide dismutase activity but decreased peroxidase levels and had no effect on catalase

These hepatic pathological alterations were associated with significantly elevated plasma IL-6 concentrations and TNF levels in both plasma and liver tissues (Fig