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Knockout of Krppel-like factor 14 (KLF14) and Krppel-like factor 15 (KLF15) in renal tubular cells, as well as activation of activating transcription factor 6 (ATF6) overexpression, can inhibit PPAR, reduce FAO mitochondrial activity, lead to lipid accumulation and insufficient energy supply in renal tubular cells, increase apoptosis, and ultimately cause lipotoxicity-induced RF [45,46,47]

This antioxidant and its associated enzymes can efficiently bind a wide range of toxins , including reactive oxygen species, peroxides, lipid peroxides, and heavy metals

MiRNA downregulation was mainly due to the hypermethylation of the CpG island in the promoter region of these miRNAs

The controversial role of human gut Lachnospiraceae