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While Lip-1 alleviated the lethality of DEM (Figure 1E), it did not prevent the fall in glutathione that was induced by aging (as assayed on Day 4) or by DEM (Figure 1F)

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This pathway, termed TMED10-channeled unconventional protein secretion (THU), relies on the ERGIC-localized TMED10 protein to facilitate the entry of IL1 family factors into the secretory system, initiating unconventional secretion 17