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In women, high levels of apoC-III were significantly and independently associated with impaired catabolism of VLDL, resulting in plasma HTG 34 , but the main effect may be due to the ability of apoC-III to inhibit hepatic TRL or remnants uptake 35

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Adenosine A1 receptor activation mediates the developmental shift at layer 5 pyramidal cell synapses and is a determinant of mature synaptic strength

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