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The major pharmacological mechanisms include the promotion of pro-survival and antiapoptotic signals [133,134], activation of growth factor signaling and inhibition of inflammatory response [135], increased insulin secretion and antioxidant molecules [136], suppression of extracellular matrix gene expression [137], and regulation lipid profiles [138]

10.1128/AAC.00395-15 109 LeeB

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The administration of Fer-1 markedly prevented ultrastructural and morphological alterations in Raji cells, such as increased density of mitochondrial membranes and incomplete mitochondrial cristae, in Raji cells (Fig