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In conclusion, targeting neutrophil-driven oxidative stress and inflammation cascades, developing nano-antioxidant-based therapies combined with anti-inflammatory interventions, offers new approaches and effective methods for clinical prevention and treatment of liver reperfusion injury

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Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF