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While some patients notice brighter and more even-toned skin, results are not guaranteed, and scientific evidence regarding long-term skin-lightening effects remains limited

This AOP is constituted the MIE as sterol regulatory element binding transcription factor 2 (SREBF2) gene activation and repressions of glutathione synthetase (GSS) and glutathione S-transferases (GSTA1, GSTA2, GSTA3, GSTA5) genes and the KEs as perturbation of cholesterol, though up regulated unsaturated fatty acid concentration, and glutathione homeostasis leading to oxidative stress, DNA damage and AO as hepatotoxicity (HepG2 cell death)

Improvements in pain intensity and frequency, with a minimum improvement of 50%, indicate successful treatment [7]

2018 Mar 1;16(1):113-24