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Studies have shown that in patients with AP, the degree of glucose metabolic disturbances is positively correlated with the intensity of the inflammatory response ( In conclusion, hyperglycemia, IR, abnormal insulin secretion from pancreatic -cells, and glucose metabolic disturbances are closely interrelated and interact synergistically to exacerbate disease severity in AP

HFD + L-NAME mice also developed a systemic inflammatory state and concomitant activation of iNOS, paralleling findings in human HFpEF hearts and suggesting iNOS as a key mediator of HFpEF development (Schiattarella et al., 2019)