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Conclusion BsADCs are a transformative therapeutic modality for breast cancer

Iron can only be released after reduction to its ferrous form (Fe 2+ ), a process facilitated by superoxide radicals, acidic pH, and catecholamines, conditions prevalent during hypoxia and ischemia (48, 49)

Further investigations are warranted to fully explore how LSC3 and the redox pathways can be exploited for the treatment of LUAD and other cancers, especially for those with poor outcomes and limited therapeutic options

238 In PH, neointima formation involves a Notch3-marked subset of SMCs and inhibition of the Notch signaling has been shown to improve inflammation-driven mPAP