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Specifically, the control group and model group were distinctly separated along the PC1 axis, indicating that the model induction caused significant metabolic perturbations

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Storage Refrigerate 28 C after reconstitution

Results: Seladelpar treatment resulted in a profound remodeling of the dysregulated proteome of PBC-like cholangiocytes (H69-miR-506) compared with control cells (178 upregulated and 91 downregulated)