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The c-Jun NH 2 -terminal kinase (JNK) pathway is activated by OS and cytokines (tumor necrosis factor- and FAS) and, similar to the ERK pathway, stimulates a cascade of phosphorylation reactions involving MAPKKK, MAPKK, and ultimately phosphorylating JNK on threonine and tyrosine residues

This is concerning as SLE patients may already be in a hypercoagulable state, and development of pulmonary emboli (PE) and deep vein thromboses (DVT) are common complications of COVID-19 disease ( in vitro , are strong procoagulants in vivo

500,501,502 To conclude, leveraging ZIP2 to modulate cellular zinc metabolism could offer an innovative approach for treating these two diseases

Some patients may switch between tirzepatide and semaglutide under medical guidance