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To receive statistically correct data, the number of measured RBCs during one experiment was 250700 in control and 150500 under the action of oxidative stress in 910 different channels in the same device

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Since BSO treatment appears to be critical for FAC-dependent cell death (Figure 1), it can be assumed that mitochondrial LPO is not sufficient for ferroptosis

Therefore, the relationship between their mechanisms of hypoglycemia, organ protection, and regulation of Cu metabolism is widely studied