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Allele loss mutation screen at the Peutz-Jeghers (LKB1) locus (19p13.3) in sporadic ovarian cancers

Dutta et al

Normally, AECs exhibit sophisticated and efficient repair mechanisms, with wound healing progressing through four distinct stages: coagulation/clotting, inflammatory cell migration, fibroblasts migration/proliferation/activation, and tissue remodeling and degradation ( Complex bidirectional interactions between epithelial cells, mesenchymal cells, and ECM subsequently induce a series of cellular events, including fibroblasts differentiation into myofibroblasts, migration, proliferation, and activation of AECs and fibroblasts, inhibition of apoptosis, and persistent abnormal ECM secretion

The results show that an NO adduct on the sulfhydryl group effectively drives intermolecular disulfide bond formation in TTR, converting native protein into the dsNNTTR species, see Fig