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doi: 10.1167/iovs.66.5.23

Glp-2: a poorly understood mediator enrolled in various bariatric/metabolic surgery-related pathophysiologic mechanisms

This pathway activation enables: Accelerated migration of repair cells to injury sites Improved cell survival under oxidative stress conditions Enhanced F-actin formation essential for cellular movement Increased fibroblast outgrowth and spreading capacity Research using FITC-phalloidin staining demonstrated that BPC-157 potently stimulates F-actin formation, providing the structural foundation for improved cellular mechanics throughout the healing process

Cells containing the library members are propagated in the absence of selection for the selectable marker for at least several generations, after which selective pressure is added