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Under normal conditions, NRF2 is degraded by its negative regulator KEAP1 via the ubiquitin-proteasome pathway [10]

View Study PCO-02 Safety and Pharmacokinetics Trial (Phase 1)

Given these biological and methodological constraints, the field is increasingly turning toward multimodal strategies to capture the complexity of neuroprotection

To compensate for this sudden loss of blood pressure, the autonomic nervous system triggers severe tachycardia (a rapid heart rate), manifesting as Postural Orthostatic Tachycardia Syndrome (POTS) and other forms of dysautonomia