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10.1080/1744666X.2016.1221762 [DOI] [PubMed] [Google Scholar] 32.Hallock KK, Mizerak MR, Dempsey A, MacZuga S, Kirby JS

otherwise, a fixed-effects model was employed

Collectively, this damage results in decreased melanocyte pigment-forming capacity and may even induce apoptosis through activation of inflammatory pathways such as the MAPK and p53 pathways.13 Saxena et al

While the N370S, R496H, E326K and the R44C mutations have been identified in ET cases [ 89 ], studies so far, regarding ET and GBA , have not provided evidence that the GBA gene is a major risk factor for ET [ 89 , 95 ]