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Dietary cuprizone-induced demyelination In this model, mice are fed with the copper chelator cuprizone, leading to oligodendrocyte death and subsequent demyelination followed by spontaneous remyelination, which is initiated during the late demyelination phase and continues robustly over the subsequent 36 weeks after withdrawal of cuprizone

1998) and doxorubicin (Dobbs et al

Methods Sixtytwo women with melasma were enrolled and randomly assigned to two groups for 12 weeks of treatment

Myo-Inositol serves as a well-established glial marker due to its preferential uptake by astrocytes and its role in maintaining cerebral osmotic homeostasis, while also being associated with microglial activity (Solomonia et al., 2013)