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Inhibition of interleukin-6 abolishes the promoting effects of pair housing on post-stroke neurogenesis

Until recently, the majority of FDA-approved AD drugs on the market include cholinesterase inhibitors and a single NMDA antagonist, which solely tackle the symptoms of AD and neglect the underlying mechanisms of the disease (Wang and Reddy, 2017

Cuproptosis increases the stability of NFE2L2 by suppressing its proteasomal degradation, leading to transcriptional upregulation of GCLC and GCLM and enhanced GSH synthesis, thereby suppressing cuproptosis in pancreatic cancer cells 169