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Moreover, we confirmed that overexpression of sVLDLR further suppressed TGF-induced p-Smad2/3 in either LRP5 or LRP6 KO cells, indicating that VLDLR plays an anti-TGF signaling role that is independent of Wnt coreceptors

Abstract T cell metabolism is central to cell proliferation, survival, differentiation, and aberrations have been linked to the pathophysiology of systemic autoimmune diseases