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Additionally, TNBC cells may activate compensatory signaling pathways, including PI3K/AKT and MAPK/ERK, and exhibit epigenetic plasticity, which allow them to adapt and evade therapy

On the other hand, the abilities of different angiotensin-converting enzyme inhibitors (ACEis) captopril (a thiol ACEi), enalapril, and lisinopril (two nonthiol ACEi) to mitigate mitochondrial toxicity induced by PQ show that captopril is a more effective antioxidant than the nonthiol ACEi due to captoprils abilities to scavenge ROS (Ghazi-Khansari et al

DSIP had no effect on somatotrope and corticotrope differentiation

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