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In particular, our study showed that the intracellular depletion of GSH is able to induce cellular stress in NO-producing cells through a NO-dependent mechanism, such as inhibition of CcOX activity, DNA damage, and S-NO and 3-nitrotyrosine (NO 2 -Tyr) protein accumulation ( Figure 1 )

GLUT1 activity is closely regulated by its expression levels and membrane localization, both of which respond dynamically to cellular energy demand and metabolic stress

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