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Many drugs look promising in cells, mice, or tissue samples but fail in later stages because: The treatment doesnt work well enough in humans It causes side effects at real doses The effect fades over time The human joint environment is much more complex than the lab So while we are hopeful, there is a long road ahead before this approach could become a real treatment

According to Science Direct [1], GHK-Cu functions as a potent tripeptide-copper complex that modulates fibroblast activity by inducing a significant increase in collagen synthesis, specifically demonstrating up to a 70% increase in glycosaminoglycan and hydroxyproline production compared to control groups in experimental models

References Stockwell BR, Angeli JPF, Bayir H, Bush AI, Conrad M, Dixon SJ et al (2017) Ferroptosis: a regulated cell death nexus linking metabolism, redox biology, and disease

Similarly, we can also provide some insight into the potential negatives which is exactly what I am going to do here (Li, 2005