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doi: 10.1038/s41421-022-00495-y 457 WuS.SongR.LiuT.LiC

The effects of genetic polymorphisms on the rates of drug metabolism are well known, but how these polymorphisms influence the susceptibility to drug-drug interactions is less clear, in particular considering warfarin-drug interactions, whether reduced function proteins (e.g., CYP2C9*3 ) are inhibited to the same extent in vivo as wild-type proteins (e.g., CYP2C9*1 )

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