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The first case involved a 72-year-old man with refractory CD and severe neurological symptoms, including cortical myoclonus and ataxia

[83] they demonstrated, with in vitro and in vivo experiments, that pulmonary microvascular endothelial barrier dysfunction plays an important role in the mechanism by which COPD affects the progression of atherosclerosis and that oxidative stress and ferroptosis are involved in this effect

Jenica David Nallathambi, junior, Deans List, College of Arts and Sciences, biochemistry

There is evidence that these reactions occur within atherosclerotic plaques and they present the most accepted putative mechanism of a direct participation of GGT in the pathophysiology of atherosclerosis leading to promotion of atherosclerotic process, plaque instability and coronary ischemic events (89)