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For example, elevated p62/SQSTM1 can identify tumors resistant to proteasome inhibition, guiding clinicians toward autophagy-targeted strategies

SARS-CoV-2 infection: hijack of host cellular metabolic machinery The SARSCoV2 obviously lacks metabolic enzymes, a critical requisite for viral genomic replication, protein synthesis, and lipogenesis

In contrast, many proteins that are key to normal synaptic function are aberrantly S -nitrosylated in AD, which results in synaptic damage and neurodegeneration (Nakamura et al., 2013)