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Regarding the impact of NIF on MTX-induced intestinal injury, NIF exhibited potent antioxidant benefits against MTX-provoked intestinal injury by controlling PPAR-, SIRT1, and Nrf2 redox-sensitive signals expression (Abd-Alhameed et al

The heavy subunit catalyzes formation of -glutamylcysteine, whereas the light subunit increases affinity for glutamate and decreases the inhibition by glutathione (Seelig et al., 1984