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In some situations, an understanding of the mechanism of toxicity has allowed the development of appropriate treatments or antidotes

i.p.) inhibits acute and relapsing EAE (experimental autoimmune encephalomyelitis) induced by proteolipid protein PLP p139-151, reduces lymphocytes proliferation, results downregulation of interleukin (IL)-17 and a marked decrease of the IFN-/IL-4 ratio [6]

S molecules in the cis-Golgi with an inaccessible extended motif will be incompetent for coatomer binding and continue anterograde export to the PM

Antibody responses induced by long-term vaccination with an octovalent conjugate Pseudomonas aeruginosa vaccine in children with cystic fibrosis