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Overcoming these challenges requires spatiotemporally dynamic regulatory strategies: maintaining appropriate ammonia levels during early treatment to promote T cell function, while controlling toxicity in later stages through lysosomal pH modulators (e.g., C381) or urea cycle enzyme activators

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ISCA2 serves as a critical mediator of Fe-S cluster biogenesis and contributes significantly to the development of multiple mitochondrial dysfunction syndromes (Weiler et al., 2020