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The reactivity of GSH towards reactive oxygen species (ROS) is marginal and, in fact, the antioxidant action is largely accomplished by donating electrons to glutathione peroxidase (GPX) [25]

A likely generalization follows the association of models of chemically induced inflammation (trinitrobenzenesulfonic acid and dextran sodium sulfate) with a marked reduction in blood flow[17-19]

Thus, by contradiction, the survivors at high temperatures are not heat-tolerant mutants or persistor-like cells

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