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Experimental data therefore suggest that pharmacological strategies preserving or re-balancing mitochondrial function may normalize several inter-organellar circuits simultaneously, rather than acting on a single linear pathway such as oxidative stress or necrosis

Research by Bin Li reveals that SAL activates the AMPK pathway by suppressing miR-802, upregulating p-ACC, CPT-1, and PPAR protein expression while downregulating SREBP-1c, consequently reducing hepatic TG, TC, and LDL-C levels while increasing HDL-C in NAFLD mice, thus modulating lipid metabolism to exert protective effects against NAFLD (Li B

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Temporal study of acetaminophen (APAP) and S-adenosyl-L-methionine (SAMe) effects on subcellular hepatic SAMe levels and methionine adenosyltransferase (MAT) expression and activity