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Notably, the increased expression of the eNOS gene reduces the expression of adhesion molecules in endothelial cells, mitigates colitis induced by DSS in mice, and is associated with severe cases of UC, as documented (145)

Moreover, FA improves the anti-inflammatory and antioxidant capacity of UC, which is also closely related to its ability to suppress the TLR4/NF-B and NF-B/iNOS/NO pathways

Mechanisms such as microembolism, neurohormonal activation, exogenous and endogenous toxins, hemodynamics, inflammatory factors, I/R injury, and OS may be involved

When a virus infects a cell, it produces damage-associated molecular patterns (DAMPs) and pathogen-associated molecular patterns (PAMPs), which could stimulate antiviral reaction responses through neighboring cells and attract innate and adaptive immune cells such as macrophages, natural killer (NK) cells, and gamma delta T (T) cells