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Another study reported that hemin-induced BBB damage and neuropathy showed intracellular Fe 2+ accumulation in endothelial cells and caused pericytes-induced endothelial barrier dysfunction

It has been found that the endoplasmic reticulum forms a separate cellular compartment from the cytosol, having a relatively oxidized state as compared to the cytosol, and thereby promoting the formation of disulfide links in proteins, which are often necessary for normal activity