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EDC and NHS were added onto rGO, with the molar ratio of rGO:EDC:NHS as 40:50:73

Given the role of mutant-p53 in ROS production through induction of NOX4 and NOX4 overexpression in RA-FLSs, it seems that impaired function of p53 contributes to the overproduction of ROS in RA synovium which can leads to new mutations in other genes and RA development (Fig