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We also determined thiol levels in the PfK13 knocksideways line in the presence of RAP and found that mislocalisation of PfK13 significantly elevated levels of reduced GSH and its precursor gGlu-Cys (Fig 4D)

The finding that the injection of GSH itself did not blunt the ventilatory depressant effects of fentanyl raises the tentative suggestion that the bioactivity of GSHee may be due to the enhanced ability of the thiolester to enter cells that control breathing and that this intracellular entry is related to the sustained efficacy of GSHee against some of the actions of fentanyl

The pathogenicity and synergistic action of Th1 and Th17 cells in inflammatory bowel diseases