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Abstract Glutathionylation is an important Phase II biotransformation pathway that utilizes glutathione transferases (GSTs) to conjugate glutathione with reactive electrophiles represented by a great diversity in chemical structures

1 Probenecid, commonly used in the treatment of gout, has been shown to extend the half-life of acetaminophen, and it has been suggested that the maximum daily dose of acetaminophen should be reduced to 3g in patients concurrently taking probenecid

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This includes hormone therapies, peptides, and some biologics