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Mechanistically, aberrant copper homeostasis during I/R impairs Fe-S cluster integrity, increases ROS production, and depletes GSH, culminating in synergistic proteotoxic and oxidative damage [160]

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The tripeptide itself has documented gene regulatory activity it upregulates over 30 genes involved in tissue remodeling, anti-aging, and repair, while downregulating pathways linked to inflammation and cancer progression

When telomeres shorten, cells are more likely to become senescent, linking TERT and FOXO4-DRI through shared longevity pathways