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The mechanism underlying these opposing gastrointestinal effects likely relates to semaglutides complex effects on gastrointestinal motility, with some patients experiencing accelerated intestinal transit while others experience overall slowing of gastrointestinal motility

This variation has been associated with reduced activity-dependent release of brain-derived neurotrophic factor, a protein that plays a critical role in synaptic plasticity, neuronal survival, and the formation of new neural connections

Altered gut microbiome and autism like behavior are associated with parental high salt diet in male mice

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