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DFA1, a deferric amine compound, exhibits potent chelation and ferroptosis suppression in vivo and in vitro [36]

Percent cytotoxicity was calculated as follows: (A490 of experimental culture medium-A490 of normal medium)/(A490 of maximum LDH release-A490 of normal wells) 100%

The alterations in substrate metabolism during heart failure, whether they result from the disease or represent residual compensatory responses within the body, remain controversial