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The PTEN-induced putative protein kinase 1/E3 ubiquitin ligase PARK2 (PINK1/Parkin) system plays a significant role in mitochondrial non-receptor-mediated autophagy (Pickles et al., 2018)

Such studies must be interpreted as model-specific research findings, not as human or veterinary use guidance

However, KYNA influences AMPAR activity predominantly through indirect mechanisms, namely its non-competitive allosteric inhibition of presynaptic 7nAChRs on glutamatergic terminals in mood-related circuits, leading to reduced glutamate release and downstream modulation of excitatory signalling [29, 30]