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The bioavailability was limited and was 7.78% in rats

Ninkuu et al., 2025)

Due to the very low expression of GRX1 in T cells, which does not upregulate upon activation, the GSH/GRX system cannot compensate for the loss of the TRX1 pathway, making TRX1 the sole electron donor for dNTP production in proliferating T cells