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Mechanisms 1 and 2 represent hydride gain, in which the molecule gains what amounts to be one hydride ion

parkeri survival in macrophages as compared to epithelial cells and protects them from a non-bacteriolytic, inflammasome-independent mechanism of restriction

NMDA was used to induce acute injury in primary rat cortical neurons, which markedly increased HTT mRNA expression within a short time, as well as increased the expression of caspase-6 , caspase-3 , and caspase-8 [92]

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