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doi: 10.1128/mbio.03251-21 Summary Keywords ALS, copper, protein, aggregate, TDP-43, SOD1, C9ORF72, neurodegeneration Citation Min J-H, Sarlus H and Harris RA (2024) Copper toxicity and deficiency: the vicious cycle at the core of protein aggregation in ALS

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It is hoped that in future research, further breakthroughs can be made in understanding the regulation mechanism of ferroptosis and utilizing ferroptosis to promote treatments for kidney diseases, such as acute kidney injury(AKI), chronic kidney disease (CKD), diabetic nephropathy(DN), and renal cell carcinoma

These results suggest that decreased GPX4 expression decreases cellular tolerance to external oxidative stimuli, leading to EBV reactivation