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Variability in supplementation dose and duration, concentration of bioactive compounds, participant characteristics, exercise tests and statistical considerations may help explain these disparate findings

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Similarly, HDAC3 also represses genes involved in DNA damage response and cell cycle checkpoints, enabling AML cells to evade genotoxic stress induced by chemotherapy ( In summary, dysregulated histone deacetylation establishes an epigenetic environment conducive to AML progression and therapeutic resistance

The progression time of this type of anemia to clinical B12 deficiency is 2-5 years (Vaqar S, 2023)